Enteritis : the causes and mechanisms of development

Enteritis : the causes and mechanisms of development
Launchers factors of inflammation in the intestines.

Enteritis can be acute or chronic, respectively, differ in their causes and mechanism of development.

causes and mechanism of acute enteritis

  • specific infectious enteritis caused typhoid bacteria, paratyphoid, opportunistic organisms - Proteus, E. coli and other
  • infectious nonspecific enteritis occurs when flu, sore throat;
  • parasitic enteritis (giardia, worms);
  • toxic enteritis in case of poisoning with caustic alkalis, acids, lead, mercury, arsenic, nicotine, etc .;
  • secondary enteritis (after skin burns, frostbite, tissue damage, injuries, this is due to the formation in the affected tissues of a large amount of histamine-like substances);
  • allergic and drug enteritis (iodine, salicylates, papaverine).

role of food factor in the development of chronic enteritis

Long term dieting with severe restriction of proteins, fats and carbohydrates can cause a predominance of heavy fermentation enteritis.It is also very bad for long-term use of ab

undant fatty foods in low protein content in it.

constant use of coarse plant not entirely benign chronic food injures the mucosa of the small intestine, reducing its protective barrier function.

enteritis Development promotes the weakening of the secretory function of the pancreas, liver and gallbladder, as well as the presence of gastritis with low acidity, a condition after resection of the stomach, the defects of the masticatory apparatus.This is due to the fact that when there is insufficient secretion of digestive juices is significantly reduced or disappears completely their bactericidal function.At the same time insufficiently digested, badly crushed and disarmed by microbial food enters the small intestine and becomes a source of inflammatory processes in it.

often observed enteritis summer as during the heat of people drink very large amounts of fluid, which causes an increase in intestinal peristalsis.His mucous becomes easily vulnerable, and even minor food or bacterial irritation contributes to inflammation.

In chronic enteritis often disturbed intestinal motility, often developing dysbiosis, and this contributes to throw the feces from the colon into the small intestine, and serves as an additional source of her infection.

enteritis in development are important predisposing factors: the general weakening of the body, the previous and accompanying diseases, surgery, stress, etc.

result is the congestion, swelling of the mucous membrane of the small intestine, the appearance of her point hemorrhages.In debilitated, malnourished patients and in severe flow process may cause ulceration, which can lead to bowel perforation and peritonitis.

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